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Transport and inhibition mechanism of the human SGLT2-MAP17 glucose transporter

Due to a shortage of oxaloacetate (resulting from limited glucose metabolism in the Krebs cycle), excess acetyl-CoA is redirected to the ketogenesis pathway in the mitochondria

NCGC is an ultra-high-throughput screening center that generates chemical probes of gene and cell functions in health and disease, and catalyzes for drug development for neglected rare and orphan diseases

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Metabolism 40 , 11851190, doi:10.1016/0026-0495(91)90214-H (1991)