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Nrf2 transcriptionally regulates FTH1 (163, 164), and interventions like resveratrol or HO-1 activation reinforce the Nrf2/FTH1/GPX4 axis to suppress ferroptosis (165, 166)

such response activates CD4 and CD8 T cells, producing tumor cell apoptosis, indicating that the TH1 or M1 response is antitumor or protective in nature

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After hypoxia, mitochondrial suspensions were isolated from the brains of these rats using the same technique as described above for polarographic measurements [2]

Under normal conditions, Nrf2 is sequestered in the cytoplasm by its inhibitor protein, Kelch-like ECH-associated protein 1 (Keap1)