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USP7 has been reported to affect the stability of HO-1 by regulating the ubiquitination of HO-1

Even mild injury prevents PT redifferentiation, promoting inflammatory PT cell aggregation and maladaptive repair via pro-fibrotic signals, extending ferroptosiss role beyond cell death to non-lethal pathological cell state regulation

In contrast, FoB exhibit lower intracellular ROS and maintain a metabolic setup that is distinct from that in MZB

This pathway, in turn, plays a vital role in generating ribose-5-phosphate for nucleotide synthesis and NADPH for reductive biosynthesis and detoxification of reactive oxygen species

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Because they work through entirely different biological pathways, there is no mechanism conflict between the two therapies