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In line with this, previous studies have shown that systemic -secretase inhibitor treatment prevents the release of A peptides into the extracellular space 51 , leads to a rapid decrease of total 30,52 and oligomeric 10 A levels in mouse models of -amyloidosis and, concomitantly, normalized neuronal activity 10

Alix-Panabires, C., Cayrefourcq, L., Mazard, T., Maudelonde, T., Assenat, E

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4a) G6PD is commonly overexpressed in many myeloid and lymphoid malignancies leading to improved proliferation, but this effect can be abrogated by pharmacologic inhibition of G6PD 87

This process, using glutamate (Glu), cysteine (Cys), and glycine (Gly) as raw materials, depends on glutamate cysteine ligase (GCL) and glutathione synthetase (GS) catalysis, present only in the cytoplasmic matrix [23]

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