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This leaves the mitochondria vulnerable to oxidative damage, directly impairing their ability to produce ATP

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In response to neuronal activity, adaptive myelination regulates oligodendrocyte lineage dynamics via Neuregulin-ErbB and BDNF pathways, NMDA-receptor and GABAergic signaling, and activity-dependent ATP-LIF transduction, ultimately reshaping myelin sheath biogenesis and architecture (Bechler et al., 2018)

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