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Mechanism 1: Upregulation of Growth Hormone Receptors (GHRs) This is the most critical mechanism for chronic tendinopathy

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While GLP-1 peptides activate only the GLP-1 receptor, Retatrutide engages GLP-1R, GIPR, and GCGR simultaneously

Research protocols sometimes combine matrikine signals with broader gene modulators to address both the targeted collagen induction pathway and the broader tissue remodeling network simultaneously

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