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Yuan et al., 2018), both of which are common free oxygen radicals with lipotoxicity ( APS could activate the nuclear erythroid-derived 2-related factor 2 (NRF2) pathway and its downstream protein glutamate-cysteine ligase and heme oxygenase-1 (HO-1) to anticipate anti-oxidative stress, reversing the marker enzyme of peroxidase catalase, MDA, SOD, and GSH-Px after suffering OS damage (Yan Y

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it is unknown whether these radical-trapping antioxidant pathways compensate for GPX4 deficiency in diabetic kidneys or if their dysfunction contributes to ferroptosis susceptibility

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