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[PubMed: 31250122] 144

The dysregulation of these pathways accumulates, ultimately causing decreased glucose uptake, impaired glycogen synthesis, and enhanced gluconeogenesis, forming the core pathological basis of metabolic disorders such as T2DM

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Structural requirements for biological activity of glucagon-like peptide-I

CNS Satiety Circuit Desensitization A poorly characterized aspect of non-response biology is what happens to central circuit sensitivity during prolonged GLP-1 RA treatment