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The GPR30 receptor agonist G1 inhibits ferroptosis and significantly improves chondrocyte viability and motor ability in mice through the YAP1/FTH1 pathway, while GPR30 receptor antagonist G15 has the opposite effect, suggesting that hormone-related ferroptosis may emerge as a promising target for alleviating osteoarthritis in postmenopausal women

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This approach ensures that the final product closely matches user expectations and, in subsequent user evaluations, is more likely to be significantly preferred over alternatives

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doi: 10.1210/jcem-24-10-1076 4 PerleyMJKipnisDM

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