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doi:10.1067/mhj.2000.107548

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Many treatment strategies aiming to directly disrupt the formation of amyloid (A) plaques and neurofibrillary tangles (NFTs), the two major AD hallmarks, showed modest or no benefit [2]

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Proton pump inhibitor and histamine 2 receptor antagonist use and vitamin B12 deficiency

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EGCG has been shown to inhibit the activity of the NF-B transcription factor in various human cancer cells (Peng et al

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