J Neurochem 78:13891399 Crack PJ, Taylor JM, Ali U, Mansell A, Hertzog PJ (2006) Potential contribution of NF-B in neuronal cell death in the glutathione peroxidase-1 knockout mouse in response to ischemia-reperfusion injury
Neuron 109(3):461472 Baraboi ED et al (2010) Lesions of area postrema and subfornical organ alter exendin-4-induced brain activation without preventing the hypophagic effect of the GLP-1 receptor agonist
Increased BBB permeability permits the entry of inflammatory cytokines, LPS, and other neurotoxic chemicals that can induce or worsen neuroinflammatory responses, further affecting brain function
Stimulation of de novo glutathione synthesis by NFT concentrations in this range was observed in HepG2 cells, PHHs, or human neuronal cells, but not to the same extent in other cell types such as human iPSC-derived astrocytes
We also recognize that although our studies are crosssectional and reflect a static snapshot of GPX1 expressing cells in human pancreatic sections, they are nevertheless informative and provide important clues on beta cell stress
Arango NP, Brusco L, Mills Shaw KR, Chen K, Eterovic AK, Holla V, Johnson A, Litzenburger B, Khotskaya YB, Sanchez N, Bailey A, Zheng X, Horombe C, Kopetz S , Farhangfar CJ, Routbort M, Broaddus R, Bernstam EV, Mendelsohn J, Mills GB, Meric-Bernstam F
