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Rethinking cystic brosis pathology: The critical role of abnormal reduced glutathione (GSH) transport caused by CFTR mutation

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Anodal tDCS appears to induce similar effects, causing a transient increase in neuronal spontaneous discharge followed by enhanced cortical inhibition

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Return and Re-Use of Released GABA Although GABA contributes at most 1020% of fluxes in the glutamineglutamate (GABA) shuttle, GABAergic signaling is essential in the regulation of endocrine functions and in brain information processing