In this study, we found that in vitro exposure to IL-1 was sufficient to induce pT H 17 cells, driving an increase in CD38-expressing cells, whereas in vivo targeting CD38 consistently decreased cortical IL-1 in the AD brain, suggesting that CD38 treatment could lead to IL-1-mediated modulation of T H 17 cell pathogenicity
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Journal of Nutritional Biochemistry, 67(2), 75-82
ME/CFS clinician coalition, diagnosing and treating myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS)
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