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At the same time, the company decided to terminate the clinical development of another small molecule agonist, Lotiglipron
This is attributed to the loss of MT stabilization and even destabilization of MTs, and also destabilizing interactions with actin filaments, as the phosphorylation of Tau at specific sites reduces the capacity of Tau to bind MTs that contributes to neuronal structural instability and neurodegeneration (Johnson and Stoothoff, 2004
2006;47(5 Suppl 3):S11-145
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