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With respect to the latter, we have previously demonstrated that apigenin or verapamil, which stimulate ABCC1-mediated drug efflux [57], led to the selective oxidative cell death of multidrug resistant cells expressing ABCC1, in the absence of cytotoxic drugs, due to rapid efflux of intracellular glutathione and a rise in ROS levels [56]

Krishnan and A.T

HS is increasingly recognized as a disease driven by systemic inflammation and metabolic dysfunction, with evidence supporting the use of treatments that address both pathways