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Under normal conditions, the production of cytokines such as IL-6, TNF-, IL-1, and IL-8 is crucial in the recruitment of immune cells, such as neutrophils and macrophages, to the site of infection and eliminating pathogens (98, 149153)

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As the virtues lie in the middle, the activity of G6PD must remain within a narrow range to achieve maximum efficiency, and it is highly probable that the presence of a functional genetic defect in G6PD most likely perturbs this delicate balance [115]