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Similar content being viewed by others Log in or create a free account to read this content Gain free access to this article, as well as selected content from this journal and more on nature.com or Accession codes Accessions GenBank/EMBL/DDBJ References Acuna-Goycolea C, van den Pol A (2004)

Figure 3 4.3 Fatty acid metabolic dysregulation induces hyperuricemia in gout pathogenesis Hyperuricemia is an established modifiable risk factor for gouty arthritis (GA), which directly influences disease progression through dynamic fluctuations in serum urate levels ( de novo fatty acid synthesis (DNL) induce purine salvage synthesis pathways to promote excessive uric acid synthesis ( HIF-1 serves as the primary molecular mediator of the hypoxic response

cysteine as a proteinogenic amino acid and cysteine as an amino acid involved in the GSH- and thus redox- homeostasis

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Maintain a food diary to find which meals and habits exacerbate symptoms