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It is therefore suggesting that the slight change of relaxation observed in the aorta without endothelium ex vivo might be due to some mechanism unrelated to the direct nitric oxide stimulation which needs further investigation

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6a, the mice in the control group presented numerous A plaques stained by 6E10, whereas treatment with (-)-Vestitol, Salviolone, their combination, and Donepezil reduced the A plaque burden to varying extents

But again, I ask: what causes it