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[DOI] [PubMed] [Google Scholar] 43.Anttila S, Hirvonen A, Vainio H, Husgafvel-Pursiainen K, Hayes JD, Ketterer B

Ethics statement The studies involving humans were approved by the Ethics Committee of Fujian Medical University Union Hospital (Approval No

That said, individual results vary

Deficiency in key autophagy molecules, such as Atg7 and Beclin-1, may contribute to DOR (Texada et al., 2019), as confirmed in animal studies

Top panels illustrate mechanisms of action: atropine (2.1 mg) competitively antagonizes acetylcholine at muscarinic receptors to reverse cholinergic effects (e.g., bronchorrhea, bronchospasm, bradycardia), while pralidoxime (2PAM, 600 mg) reactivates acetylcholinesterase by cleaving the nerve agentenzyme bond prior to irreversible aging. Middle panels depict the dualchamber autoinjector design, which contains atropine and pralidoxime in separate compartments and delivers both drugs sequentially via a springloaded intramuscular mechanism from a single needle

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