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Inhibition of SQSTM1 or ACSL4 disrupts this crosstalk, suggesting that the autophagy-ferroptosis cross-pathway represents a potential therapeutic target for dry eye disease (168)

(2018) 237:R117

Several research groups have focused on the mechanism that underlying the features of tumorigenesis and cancer cell survival in the setting of SDH mutations

Harvard Health Publishing

By mapping the cascade from zinc imbalance to molecular dysfunction, organelle stress and disease expression, the framework provides an early overview that anchors the detailed sections that follow and highlights mechanistic points of intervention with potential relevance for future therapeutic modulation 18,19,20,21

References James, C