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International Journal of Tissue Reactions

In: Ishaaya I, editor

Beyond proteasomal targeting, non-canonical ubiquitin modifications, including K63-linked polyubiquitination, regulate signaling, endocytosis, kinase activity, and DNA damage responses, while mono- and multi-ubiquitination influence chromatin dynamics, DNA repair, and subcellular localization (Fig

The majority of data supports the notion that the most likely sources of transmission are person-to-person contact in families and/or exposure to a common source of infection such as contaminated water or food (14-16)

SGLT2 inhibition modulates NLRP3 inflammasome activity via ketones and insulin in diabetes with cardiovascular disease

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