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Synaptic plasticity and aging [edit] As the brain ages, many individuals experience a decline in memory and learning, which has been known to be linked to changes in synaptic plasticity

First and most importantly, as an observational study, it cannot determine a causal association between variables

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In OA models, ACSL4 inhibition dramatically decreases ferroptosis-associated lipid peroxidation ( in vitro models, inhibiting cartilage chondroptosis and improving OA ( Excessive lipid metabolic reprogramming in OA significantly raises ferroptosis risk, so targeting lipid synthesis, transport, and degradation represents a promising upstream approach to reducing ferroptotic damage