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Reuptake and Metabolic Inhibition : Ephedrine prolongs the residency of norepinephrine in the synaptic cleft by weakly inhibiting NET-mediated reuptake and demonstrating a weak inhibitory effect on mitochondrial monoamine oxidase (MAO), the primary enzyme responsible for catecholamine degradation (Statler et al, 2023)

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Semaglutide addresses this by: Increasing Insulin Secretion: Activates GLP-1 receptors, stimulating insulin release only when blood sugar is elevated, preventing hypoglycemia

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