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GLP-1 receptor agonists also slow gastric emptying, reduce appetite through central nervous system pathways, and promote satiety, leading to reduced caloric intake and weight loss

Excess aldosterone, in addition to increasing tubular sodium reabsorption, activates inflammatory and pro-fibrotic pathways and promotes vascular remodelling [15]

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When long term stress occurs, unlike other physiologic functions (homeostatic), the stress response often stays up-regulated even after the stressful situation is resolved

This issue suggests that changes in energy expenditure due to altered thyroid function may be a primary factor leading to an increase in body weight, even with normal feedback regulation [59]