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The mechanisms underlying epileptic pathogenesis and neuronal death remain unclear

The changes in the glycan of placental villous, caused by excessive ROS, can lead to the decreased level of Gal-9, a type of galectins most expressed at the maternal fetal interface, whose increased levels indicate that the placental cells are susceptible to apoptosis (93)

The net effect is dose-dependent: low doses of MB tend to improve mitochondrial function and cellular energy, whereas excessive doses may impair these processes

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Int J Biochem Cell Biol 34: 838-854

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