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Notably, studies in mice demonstrate that GLP-1R agonists specifically localize to and require arginine vasopressin neurons in the PVN to exert their hypophagic and weight-reducing effects, a process that is amplified by GIPR co-activation [81]

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First, in pivotal trials of GLP-1 RAs, there is no acute decrease in GFR that would be expected with proximal natriuresis, suggesting that there is minimal renal hemodynamic response to GLP-1 RAs

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