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Arbutin: This is a milder pigment inhibitor than hydroquinone

The microenvironment of the injured spinal cord is characterized by mitochondrial metabolic imbalances, leading to the excessive production of free radicals and the consequent peroxidation of cell membrane lipids, which results in spinal cord nerve cell damage and even death [3,4,5]

Zheng H, Jiang J, Xu S, Liu W, Xie Q, Cai X, Zhang J, Liu S, Li R (2021) Nanoparticle-induced ferroptosis: detection methods, mechanisms and applications

Braun, L

Cellular senescence as a possible mechanism for halting progression of keloid lesions

Thus, by contradiction, the survivors at high temperatures are not heat-tolerant mutants or persistor-like cells