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Patients were censored when the time window ended or the day after the last fact in their record
Hard to mess up
Rodent studies suggest that GLP1RA administration to the dam in pregnancy alters fetal growth, and GLP1RA administration directly to neonates alters development of the hypothalamus
The preferential fat loss likely results from glucagon receptor activation, which increases resting energy expenditure and promotes fat oxidation as the primary fuel source, sparing protein catabolism for energy needs
Fetal programming is associated with placental dysfunction, which is mediated trough gene and environment interactions and can be seen in conditions such as intrauterine growth restriction [70]
Thats a red flag for metabolism, strength, and long-term weight management
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