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Our data thus far show that 5FU drives increased mitochondrial biogenesis to address a functional requirement for increased mitochondrial ETC metabolism

Since its identification in 2012, ferroptosis has emerged as a key player in a wide spectrum of pathological conditions, including neurodegenerative diseases, ischemiareperfusion injury, metabolic disorders, and malignancies [2, 3]

In contrast, the survival of mice expressing a hHPN RS was similar to that of the vector-control group (AAV-EGFP), with all mice succumbing within approximately 60 h (Fig

Third, oxidative stress and mitochondrial impairment in COPD could enhance neurodegenerative processes, extending further the link between respiratory dysfunction and cerebral pathology

Streptozotocininduced diabetic models in mice and rats

[DOI] [PubMed] [Google Scholar] 67.Cover C., Mansouri A., Knight T.R., Bajt M.L., Lemasters J.J., Pessayre D., Jaeschke H