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A lysosome-to-nucleus signalling mechanism senses and regulates the lysosome via mTOR and TFEB

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Tan EK et al

But they work through different pathways, target different outcomes, and suit different patient profiles

There is overwhelming evidence that demonstrates that elevated calcium concentration, inflammation, increased oxidative stress, as well as a reduction in blood flow, are some of the pathophysiological mechanisms underlying NIHL (Figure 1

ATF4 functions as a central regulator of amino acid metabolism and is essential for the induction of serineglycine biosynthetic enzymes