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We further demonstrated that GPX4 interacts with the TAK1-TAB1/TAB3 complex, regulates TAK1 kinase activity, and activates the downstream MAPK-JNK and NFB pathways

FoxO1-deficient -cells lose their identity and revert to pancreatic endocrine progenitors characterized by expressing high levels of neurogenin-3 (Neurog3 high ), and no insulin and MafA expression 22