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It is a clinical syndrome with at least three distinct underlying mechanisms that frequently overlap in the same patient

Erbin inhibits the cell proliferation of AML cells by decreasing the phosphorylation levels of the RAS/RAF/MEK/ERK pathway (Zheng et al., 2019)

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Even though it is possible that CD38 might directly affect the brain, targeting CD38 abrogated disease-associated T H 17 cell immunity, potentially by breaking meningeal to cortical communication via modulation of IL-17A/IL-1 crosstalk (Fig

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