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GHK (glycyl-L-histidyl-L-lysine) peptide has been shown to positively influence gene expression by regulating various cellular pathways associated with tissue repair, wound healing, and anti-aging mechanisms [7], and thus has potential as an anti-fibrotic agent

136 In a UC rat model, pretreatment of bone marrow MSCs with IL-6 increased their ability to inhibit inflammatory factors

The EMPA-REG and CANVAS trials showed that the SGLT-2i empagliflozin and SGLT-1/2i canagliflozin reduced the risk of MACE in T2D patients with or at high risk of CVD by 14%, while the SGLT-2i dapagliflozin did not significantly reduce the risk of MACE, possibly due to the difference in population (approximately 60% with no established ASCVD) between previous trials and strict exclusion criteria

Nair AB, Jacob S