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Unlike many cofactors that directly participate in redox reactions, zinc's biological role largely involves structural stabilization and catalytic activity, making it indispensable in processes such as transcription, signal transduction, immune function, and apoptosis [5] (Tables 1 and 2)

(1988), published in FEBS Letters, demonstrated that GHK-Cu significantly stimulated collagen synthesis, proteoglycan production, angiogenesis, and wound closure across multiple animal and cell models

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