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Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours
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Recent investigations have unearthed a novel player in the ferroptosis domain, MBOAT2, functioning autonomously of GPX4 and FSP1, overseeing ferroptosis surveillance through phospholipid remodeling and proficiently impeding ferroptotic processes
doi:10.1007/s11356-019- 06632-7 [32] Tchounwou PB, Yedjou CG, Patlolla AK, Sutton DJ
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