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-carotene could serve as a potential therapeutic molecule for treating neuroinflammation and apoptosis in ALS patients (Krishnaraj et al., 2016) (Table 1)
As expected, silencing YTHDF1 decreased the protein abundance of SLC7A5 in KGN cells without affecting its RNA level (Fig
Rashmi Sarkar, pooled five randomised controlled trials plus one open-label study on oral glutathione
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