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All of these studies are hypothesis-generating and await confirmation in RCTs. According to Lane, the rationale for GLP-1 receptor agonists in lupus stems from the drugs demonstrating anti-inflammatory properties, potentially by inhibiting the NF-B pathway, which plays a crucial role in inflammation

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323 The lncRNA NEAT1 is highly expressed in UC patients, and in vitro experiments have shown that it regulates its downstream target LDHA by adsorbing miR-410-3p, thereby inhibiting the glycolysis pathway and improving intestinal epithelial cell dysfunction

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