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OPCs are sensitive to neuronal activity due to the expression of ion channels and neurotransmitter receptors

33 Additionally, FUNDC1, a mitophagy receptor, interacts with LC3 under hypoxic conditions, further integrating mitophagy with autophagy

Fortunately, the lung has developed adaptive mechanisms to defend itself against inhaled oxidants [4]

In this context, SLC40A1 inhibition leads to the binding of microRNA-18a-5P with RUNX1, affecting the transcription process and influencing prostate cancer cell proliferation

Taken together, our data indicate that the loss of GSH triggered by Gclc deletion induces a defective mitochondrial accumulation in FoB, which may lead to compensation through upregulation of glycolytic metabolism