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In PD cellular and animal models, treatment with rapamycin enhances autophagic activity, promotes the degradation of -syn aggregates, and reduces neuronal damage [288]

This suggests that under certain experimental conditions, IGF-1 LR3 might exhibit more pronounced anabolic actions, though these were not consistently observed across all the variables measured

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This observation is crucial as it implies that MGF may promote these reserve cells to become more actively involved in muscle formation

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Under these conditions, the copper-peptide complex remains stable and active