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Endocrinology (2014) 155:162942

Supplementary data The following are the Supplementary data to this article: References 1.Nauck M.A., Mirna A.E.A., Quast D.R

136 Other data have shown that CHO restriction is sufficient to activate the AMPKSIRT1PGC-1 network in humans even under caloric overconsumption

and cytoprotective/angiogenic effects

This is my understanding and Im hoping you can add clarity or potentially make a post addressing these concerns

Previous studies on ferroptosis have found that ferroptosis is involved in a variety of pathological processes, such as tumors [9, 10], ischemia-reperfusion injury [11, 12], and other degenerative diseases [13] associated with extensive lipid peroxidation