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In contrast, IGFBP 5 induces cellular adhesion and inhibits migration of MCF7 cells via the activation of AKT in an IGF-independent manner, consistent with previous studies suggesting an apoptotic role for IGFBP 5 in BrCa [68]

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Research also shows that IGF-1 LR3 activates the PI3K/Akt/mTOR pathway

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By controlling excess pigment activity, they promote a brighter, more even skin tone over time without surface-level bleaching