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The ensuing pathology, largely consisting of macrophage and lymphocyte infiltration of mainly the spinal cord, recapitulates the inflammatory pathology of acute MS lesions in a number of respects, but differs from MS in important ways: many EAE variants exhibit only limited demyelination confined to the spinal cord white matter, contrasting with extensive brain pathology also prominently affecting cortical gray matter in MS
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Neuroendocrinology Letters, 32(3), 273-284
Here is a more detailed look at what happens on a cellular level: First, GHK-Cu stimulates a process called angiogenesis, which is the formation of new blood vessels
It's also been shown to have potent anti-inflammatory properties, which helps create a better environment for healing to occur
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