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One of the mechanisms supporting this resistance is the interaction between FOXO4 and p53 within the nucleus

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GHK-Cu focuses on rebuilding skin structure through collagen stimulation and tissue repair, while NAD+ optimizes cellular energy and DNA repair mechanisms

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Other substituents engage in interactions with the amino acid residues of Phe357, Ser209, and Arg358 within the S2 pocket as shown in (Figure 2)