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This can be explained by the fact that the first group alone shows high leptin levels, with low ghrelin levels, possibly caused by the former
Because the hyperglycemic effect of glucagon receptor activation (hepatic glucose output) is effectively offset by the strong glucose-lowering activity of GLP-1R and GIPR co-activation (insulin secretion + glucagon suppression)
Inflammation can lead to decreased T4 to T3 conversion, increased insulin resistance, and higher estrogen levels, all of which contribute to weight gain
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