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The role of copper in this regard is argued to be protective, with copper being released into the synaptic cleft at concentrations of up to 100 M upon depolarization, inhibiting the activity of postsynaptic NMDA, AMPA, and GABA receptors upon binding, thus preventing overstimulation in a negative feedback system (DAmbrosi and Rossi, 2015)

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"Acetyl-L-carnitine in the treatment of peripheral neuropathic pain: a systematic review and meta-analysis of randomized controlled trials"