However, the mechanisms of iron overload during ferroptosis remain elusive, although several pathways have been proposed 15
Our present observations are in keeping with our previous findings (17,30,31) indicating that the production of free radical, enhanced lipid peroxidation and an impairment of antioxidizing enzyme activity, and the increased generation of cytokines, such as IL-1 and TNFalpha, contribute to the pathomechanisms of stress-induced gastric lesions
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Generally, management includes dietary modifications (low-fat, high-carbohydrate), medium-chain triglyceride (MCT) oil, and L-carnitine supplementation
Our previous studies and those of others have found that iron overload causes liver injury and liver fibrosis by inducing ROS accumulation and activating ferroptosis in hepatocytes [32]
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