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Previous mutagenesis studies showed that individual mutations of E124 3.33 , R283 6.55 , and Q120 3.29 to non-polar residues led to decreased potency of ghrelin 34,37

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K + channels, including delayed rectifier K + channels and inwardly rectifying K + channels, contribute to repolarization and recovery, regulating firing timing and patterns (Tanaka et al., 2003)