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You see, without adequate glutathione to keep free radical levels in check, these harmful molecules begin causing unprecedented levels of oxidative damage

FIGURE 8 The aforementioned abnormalities at the molecular level (downregulation of the Nrf2/HO-1 axis and GPX4) and subcellular structural level (neuronal somata and mitochondrial impairment) were consistent with the GSH metabolism perturbations identified by multi-omics analyses, collectively reflecting oxidative stress-associated damage in the hippocampus during SAE

Am J Physiol Heart Circ Physiol 2009

True recovery requires a holistic approach that includes aggressive pacing to prevent post-exertional malaise, targeted nervous system regulation, environmental detoxification, and personalized medical care

Produces a depletion of releasable neuronal monoamine neurotransmitters Similar mechanism as tetrabenazine (Austedo )

& Sherins, R