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According to the model we have previously described, K ATP current activation driven by mitochondrial instability induces lability in the cellular action potential duration and alters the refractory period, which increases the dispersion of repolarization in the tissue and promotes reentrant arrhythmias through the formation of metabolic sinks [6, 7]

De Keyser, J., Mostert, J

Beitrag zur Lehre von der Pseudosklerose (Westphal-Strmpell)

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Feature Article: Peptide Spotlight - BPC-157 and Anti-inflammatory Mechanisms Background and Mechanism of Action BPC-157 is a stable gastric pentadecapeptide with anti-inflammatory properties that stem from multiple molecular pathways that work synergistically to modulate inflammatory responses at the cellular and tissue levels

10.1016/j.jff.2020.104279 J Funct Foods