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Short-acting drugs primarily lower postprandial blood glucose levels by slowing gastric emptying, while long-acting GLP-1 RAs are more effective at reducing fasting blood glucose levels, mainly by promoting insulin secretion and inhibiting glucagon release

Results: Evidence indicates local estrogen dominance with progesterone resistance, pervasive immune dysregulation, and oxidative stress with iron-driven ferroptosis that particularly injures granulosa cells, alongside disease-relevant genetic/epigenetic regulators and reproductive-tract/gut microbiome dysbiosis

Self-reported community protocols commonly describe once-daily dosing with on/off cycling, motivated partly by the absence of any long-term human safety data and partly by a community perception of diminishing returns or tolerance over continued use

This can make therapies that stimulate GH sound pretty enticing

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